[1]
Kubota F., Shibata N., Shiihara Y., et al.: Frontal lobe epilepsy with
secondarily generalized 3 Hz spike-waves: a case report. Clin
Electroencephalogr 1997; 28: 166–171.
[2]
So N. K.: Mesial frontal epilepsy. Epilepsia 1998; 39: 49–61.
[3]
Millan E., Abou-Khalil B., Delbeke D., et al.: Frontal Localization of
absence seizures demonstrated by ictal positron emission tomography.
Epilepsy Behav 2001; 2: 54–60.
[4]
Holmes M. D., Brown M., Tucker D. M.: Are generalized seizures
truly generalized? Evidence of localized mesial frontal and frontopolar
discharges in absence. Epilepsia 2004; 45: 1568–1579.
[5]
Craiu D., Magureanu S., van Emde Boas W.: Are absences truly
generalized seizures or partial seizures originating from or predominantly
involving the pre-motor areas? Some clinical and theoretical observations
and their implications for seizure classification. Epilepsy Res 2006; 70:
41–55.
[6]
Hirsch E., Andermann F., Chauvel P., et al.: Generalized Seizures: From
clinical phenomenology to underlying systems and networks, John
Libbey Eurotext Ltd., Montrouge, 2006.
[7]
Commission on Classification and Terminology of the International
League Against Epilepsy. Proposal for a revised classification of
epilepsies and epileptic syndromes. Epilepsia 1989; 30: 389–399.
[8]
Steriade M., Contreras D.: Spike-wave complexes and fast components
of cortically generated seizures. I. Role of neocortex and thalamus. J
Neurophysiol 1998; 80: 1439–1455.
[9]
Farwell J. R., Stuntz J. T.: Frontoparietal astrocytoma causing absence
seizures and bilaterally synchronous epileptiform discharges. Epilepsia
1984; 25: 695–698.
[10]
Lagae L., Pauwels J., Monté C. P., et al.: Frontal absences in children. Eur
J Paediatr Neurol 2001; 5: 243–251.
[11]
Leutmezer F., Lurger S., Baumgartner C.: Focal features in patients with
idiopathic generalized epilepsy. Epilepsy Res 2002; 50: 293–300.
[12]
Salek-Haddadi A., Lemieux L., Merschhemke M., Friston K. J., Duncan
J. S., Fish D. R.: Functional magnetic resonance imaging of human
absence seizures. Ann Neurol. 2003; 53: 663–667.
[13]
Duncan J. S.: Imaging idiopathic generalized epilepsy. Clin EEG Neurosci
2004; 35: 168–172.
[14]
Aghakhani Y., Bagshaw A. P., Bénar C. G., Hawco C., Andermann F.,
Dubeau F., Gotman J.: MRI activation during spike and wave discharges
in idiopathic generalized epilepsy. Brain 2004; 127: 1127–1144.
[15]
Labate A., Briellmann R. S., Abbott D. F., Waites A. B., Jackson G.
D.: Typical childhood absence seizures are associated with thalamic
activation. Epileptic Disord 2005; 7: 373–377.
[16]
Gotman J., Grova C., Bagshaw A., Kobayashi E., Aghakhani Y., Dubeau
F.: Generalized epileptic discharges show thalamo-cortical activation
and suspension of the default state of the brain. Proc Natl Acad Sci USA
2005; 102: 15236–15240.
[17]
Tenney J. R., Marshall P. C., King J. A., Ferris C. F.: fMRI of generalized
absence status epilepticus in conscious marmoset monkeys reveals
corticothalamic activation. Epilepsia 2004; 45: 1240–1247.
[18]
Morrison R. S., Dempsey E. W.: Mechanism of thalamo-cortical
augmentation and repitition. Am J Physiol 19431; 38: 297–308.
[19]
Jasper H. H., Kershman J.: Electroencephalographic classification of the
epilepsies. Arch Neurol Psychiatry 1941; 45: 903–943.
[20]
Meeren H. K., Pijn J. P., Van Luijtelaar E. L., Coenen A. M., Lopes da
Silva F. H.: Cortical focus drives widespread corticothalamic networks
during spontaneous absence seizures in rats. J. Neurosci 2002; 22:
1480–1495.
[21]
Meeren H., van Luijtelaar G., Lopes da Silva F., Coenen A.: Evolving
concepts on the pathophysiology of absence seizures: the cortical focus
theory. Arch Neurol 2005; 62: 371–376
[22]
Blumenfeld H.: Cellular and network mechanisms of spike-wave
seizures. Epilepsia 2005; 46: 21–33.
[23]
Snead O. C.: Basic mechanisms of generalized absence seizures. Ann
Neurol 1995; 37: 146–157.
[24]
Buzsáki G.: The thalamic clock: emergent network properties.
Neuroscience 1991; 41: 351–364.
[25]
Stefan H., Paulini-Ruf A., Hopfengartner R., Rampp S.: Network
characteristics of idiopathic generalized epilepsies in combined MEG/
EEG. Epilepsy Res. 2009; 85: 187–198.